Pyometra
Pyometra occurs in most cases as a sequel to chronic endometritis. As a result of the inflammation, the uterus ceases to produce or release the endogenous luteolysin.
When the CL persists there is continuous influence of progesterone and the infective process progresses. With tightly closed cervix the purulent exudates occumulates.
In small number of cases the pyometra results from an embryonic or fetal death in which the corpus luteum of pregnancy persisted with subsequent invasion by Actinomyces (Corynebacterium) pyogenes.
Infection by Trichomonas fetus. Here the uterine pus is much more copious and may attain a volume of many litres. The uterus undergoes much more distension.
Clinical Signs
- Cows with pyometra show little or no signs of ill health.
- The main reason for them being examined is the absence of cyclical activity and perhaps the presence of an intermittent vaginal discharge.
Rectal Examination
- Both the uterine horns are enlarged and distended.
- Incomplete involution of the previously gravid horn may lead to unequal enlargement of horns.
Differential Diagnosis from Normal Pregnancy
- The uterine wall is thicker than at pregnancy.
- The uterus has a more ‘doughy‘ and less vibrant feel.
- Absence of double slipping, placentomes and fetal bump.
Treatment
- Prostaglandin F2α 25 mg IM
- Synthetic prostaglandin analogue 0.5 mg IM
- Results in regression of the CL- dilation of the cervix and expulsion of the purulent fluid.
- Estradiol benzoate 3.5 mg IM.
- Causes the onset of a profuse vulval discharge in 48 hours or so. 40–60 units of oxytocin a day or so later is also helpful in hastening the expulsion of fluid.
Prognosis
If the condition is not too long standing, there is reasonable possibility for conception.
